In cases of acute intoxication with propane butane (due to damage in heating appliances where propane-butane is used as energy source for heating), asphyxia. Butane is an organic compound with the formula C4H10 that is an alkane with four carbon . It enters the blood supply and within seconds produces intoxication. Butane is the most commonly misused volatile substance in the UK, and was the. Butane is a commonly misused volatile agent, and a cause of intoxication. We present a case, who had a syncope and persistent ventricular.
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One guinea pig exposed for 2 h was examined histopathologically 7 days after exposure; no effects were found. Froth was present in the trachea and bronchi; pulmonary edema was also reported. Thus, the AEGL-2 values for min and 1, 4, and 8 h will be set equal to the 2-h concentration.
A CT head scan on the day following admission wu the hospital was normal. The physiologic responses were very briefly reported. Clear Turn Off Turn On.
Butane intoxication–three case reports.
Most of these cases involved inhalant abusers with repetitive exposure intoxicqtion butane. Concerned parents Children affected by a parent’s drinking Alcohol and Cancer Worried about someone?
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He had a history of butane abuse. Anesthetized dogs were exposed to butane by a tracheal cannula, and epinephrine 0.
For the same reasons as for AEGL-1, steady-state plasma concentrations will be reached within intoxicaiton min of exposure. Inhalation for 2 h, butane concentrations not specified. The experimental procedure in the study by Stoughton and Lamson is poorly described, but suggests that the reported concentrations of butane are probably initial concentrations in a closedchamber setting. Personal information regarding our website’s visitors, including their identity, is confidential.
In another case, a swollen brain was observed in a year-old girl without a history of butane abuse Williams and Colewhereas a CT head scan showed no abnormalities in a year-old male with a 3-year history of butane intodication Edwards and Wenstone Case Reports In nonfatal cases, butane appears to have frequently affected the heart and brain. Ventricular tachycardia and ventricular fibrillation were noted.
The mechanism of this complication is discussed and compared with the literature.
Butane – Drug and Alcohol Information and Support in Ireland –
The min value is supported by human data. If you are a subscriber, please sign in ‘My Account’ at the top right of the screen. They reported that exposure to slowly increasing concentrations of butane up to 50, ppm total exposure duration at least 10 min and a short exposure duration not specified toppm on the same day did not result in serious complaints Patty and Bugane Therefore, time extrapolation was performed from 10 min to and min exposures, where the steady-state concentration was calculated.
Butane abuse associated encephalopathy; pp. Most commercially available butane also contains a certain amount of contaminant oil which can be removed through filtration but which will otherwise leave a deposit at the point of ignition and may eventually block the uniform flow of gas.
They reported that exposure to butane at slowly increasing concentrations up to 50, ppm total exposure duration at least 10 min and toppm butaen exposure, exact duration unknown on the same day did not result in serious complaints Patty and Yant Light anesthesia can be considered serious enough to impair escape, and could be used as basis for AEGL An ambulance arrived within 5 min.
The 2-h LC 01 for mice is intoxictaion as the starting point butanee the AEGL-3 values, because it is the lowest concentration tested in what appears to be a more susceptible species. Ifkharen aM. Light anesthesia was defined as being unable to maintain an upright position in a rotating bottle 2 mice in a 2-L bottle.
Death was attributed to multiple organ failure involving the CNS brain edemacardiovascular system, pulmonary system, buutane the liver. Shugaev determined LC 50 values for butane for 2-h exposures in mice and 4-h exposures in rats. Subjects were exposed at slowly increasing concentrations up to 50, ppm continuous exposure test, total exposure was at least 10 minfollowed by exposure to fixed exposure concentrations for a short duration exact duration unknown butqne the same day intermittent exposure test.
The database for butane is poor and important studies date back to the s or s. Carr F, Prasad B. Tetracarbane never recommended . Substance abuse is one of the predominant causes of death from butane intoxication. Concentrations of hydrocarbons in tissues as a measure ingoxication toxicity. Successful resuscitation from recurrent ventricular fibrillation secondary to butane inhalation.
She collapsed when running away from the police Williams and Cole Concentrations of hydrocarbons in tissues as a measure of toxicity; pp. Most deaths attributed to butane use are caused by SSDS. Default Values in Occupational Risk Assessment; Its main uses are in the production of chemicals like ethylene and 1,3-butadiene, as a refrigerant, as an aerosol propellant, as a constituent in liquefied petroleum gas, and as the main component intooxication gas lighter refills.
No adequate human data relevant to AEGL-2 effects were found. The term may refer to either of two structural isomersn -butane or isobutane also called “methylpropane”or to a mixture of these isomers.
Animals were killed immediately after exposure.